Respond on two different days\u00a0who selected different factors than you, in one or more of the following ways:
\nShare insights on how the factor you selected impacts the pathophysiology of diabetes mellitus and diabetes insipidus.
\nOffer alternative diagnoses and prescription of treatment options for diabetes mellitus and diabetes insipidus.
\nValidate an idea with your own experience and additional research.<\/p>\n
\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0Main post
\nDiabetes affects millions of Americans and the cost of diabetes is a constant healthcare stressor as Insulin costs continue to rise. Diabetes mellitus\u00a0is separated into three classes; Type 1, the pancreas fails to produce enough insulin to meet the body\u2019s demand and Type 2, where the body\u2019s cells do not respond to the insulin production and can be managed with lifestyle modifications, and gestational diabetes. Diabetes\u00a0insipidus\u00a0is not as common as diabetes mellitus but can have a severe effect on the human body due to the fluid\u00a0imbalance that is created. It is important for the practitioner to understand the\u00a0pathophysiologies\u00a0and the roles behavior and ethnicity play in the diagnosis and treatment.\u00a0
\nPathophysiology\u00a0of Diabetes Mellitus
\nType 1 diabetes mellitus is an autoimmune disease that results from beta-cell destruction in pancreatic islets.\u00a0Beta-cell death via virus directed or physiological mechanisms induces the release of antigens and initiation of immune responses against other beta-cells\u00a0(Saberzadeh-Ardestani\u00a0et al., 2018). Type 2 diabetes also has beta-cell destruction\u00a0more but, more variable different degrees of beta-cell failure relative to varying degrees of insulin resistance.\u00a0Kumar et al.\u00a0(2018) acknowledge that\u00a0insulin resistance\u00a0impairs the ability of muscle cells to take up and store glucose and triglycerides, which results in high levels of glucose and triglycerides circulating in the blood.\u00a0Type 2 diabetes can be managed with diet and exercise; like these, both can improve insulin resistance and delay the long-term complications associated with diabetes mellitus.
\nPathophysiology\u00a0of Diabetes\u00a0Insipidus
\nDiabetes\u00a0insipidus\u00a0is separated into four classifications; central,\u00a0nephrogenic,\u00a0dipsogenic, and gestational. The results are that the body excretes an abundance of urine, causing the patient to have an unquenchable thirst.\u00a0Kalra\u00a0et al. (2016) describe central diabetes\u00a0insipidus\u00a0due to impaired secretion of arginine vasopressin\u00a0could result from traumatic brain injury, surgery, or tumors whereas\u00a0nephrogenic\u00a0diabetes\u00a0insipidus\u00a0due to failure of the kidney to respond to arginine vasopressin is usually inherited.\u00a0
\nDifferences and Similarities\u00a0of Hormonal Regulation
\nDespite sharing a name, the differences between diabetes mellitus and diabetes\u00a0insipidus\u00a0are great, from the\u00a0pathophysiology\u00a0to the treatment. Similarities between diabetes\u00a0insipidus\u00a0and diabetes mellitus due to hormonal changes lie within the\u00a0symptoms\u00a0that the patient is experiencing.\u00a0Diabetes mellitus involves blood sugar levels and insulin resistance;\u00a0diabetes\u00a0insipidus, it isn\u2019t blood sugar that is the problem, but blood water levels. The similarities of these diseases lie within the signs and symptoms that the patients may be present. Diabetes\u00a0insipidus, excessive fatigue occurs because of an overall lack of hydration or an electrolyte imbalance. For diabetes mellitus, excessive fatigue generally occurs because blood sugar levels are too low or too high.\u00a0Excessive thirst occurs in diabetes\u00a0insipidus\u00a0because the body senses a lack of Vasopressin, and so it demands more fluids because it thinks it needs them. For diabetes mellitus, excessive thirst occurs because of excessive glucose levels that need to be expelled from the body.\u00a0
\nEthnicity and Behavior\u2019s Impact\u00a0
\nResearch by\u00a0Saberzadeh-Ardestani\u00a0et al.\u00a0(2018) shows\u00a0environmental factors include reduction in gut\u00a0microbiota, obesity, early introduction to fruit or cow milk during childhood, gluten, toxins, lack of vitamins, and viruses play a role in the development of diabetes mellitus type 1; while\u00a0previously most prevalent in Europeans, it is becoming more common in other ethnic groups. Diet and\u00a0exercise\u00a0can help delay diabetes mellitus type 2 from other disease processes related to diabetes mellitus. Diabetes\u00a0insipidus\u00a0is common when a traumatic brain injury (TBI) occurs, wearing seatbelts when driving and helmets when participating in cycling or sporting events can decrease the opportunity for diabetes\u00a0insipidus\u00a0to occur.\u00a0
\nConclusion
\nIt is important for a practitioner to distinguish between the multiple types of diabetes that patients may present with, although signs and symptoms may be similar, the diagnosis and treatments are completely different. Patient education for each disease is also important because diabetes mellitus type 1 cannot be managed with diet and exercise alone. Diabetes\u00a0insipidus\u00a0can occur, but the underlying cause must be singled out to classify and treat. Central diabetes insipidus\u00a0may require\u00a0long term treatment depending on the extent of trauma, whereas gestational diabetes\u00a0insipidus\u00a0is usually a short term treatment, but the treatments remain the same. \u00a0\u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0 \u00a0<\/p>\n
\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0\u00a0References
\nKalra, S.,\u00a0Zargar, A. H., Jain, S. M.,\u00a0Sethi, B.,\u00a0Chowdhury, S., Singh, A. K., \u2026\u00a0Malve, H. (2016). Diabetes\u00a0insipidus: The other diabetes.\u00a0Indian Journal of Endocrinology & Metabolism,\u00a020(1), 9\u201321. https:\/\/doi-org.ezp.waldenulibrary.org\/10.4103\/2230-8210.172273Kumar, A. S.,\u00a0Maiya, A. G.,\u00a0Shastry, B. A.,\u00a0Vaishali, K.,\u00a0Ravishankar, N.,\u00a0Hazari, A., … &\u00a0Jadhav, R. (2018). Exercise and insulin resistance in type 2 diabetes mellitus: A systematic review and meta-analysis.\u00a0Annals of physical and rehabilitation medicine. https:\/\/doi-org.ezp.waldenulibrary.org\/10.1016\/j.rehab.2018.11.001Saberzadeh-Ardestani, B.,\u00a0Karamzadeh, R.,\u00a0Basiri, M.,\u00a0Hajizadeh-Saffar, E.,\u00a0Farhadi, A., Shapiro, A. M. J., \u2026\u00a0Baharvand, H. (2018). Type 1 Diabetes Mellitus: Cellular and Molecular\u00a0Pathophysiology\u00a0at A Glance.\u00a0Cell Journal (Yakhteh),\u00a020(3), 294\u2013301. https:\/\/doi-org.ezp.waldenulibrary.org\/10.22074\/cellj.2018.5513\u00a0<\/p>\n","protected":false},"excerpt":{"rendered":"
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